2024-01-30 08:43:00
new discovery Shaking up the medical world around the world After research reported that “Alzheimer’s disease” memory loss Can be transmitted from person to person. If hormones are received from a deceased donor pituitary gland
Summary of explanations from neurology specialist University College London Head of the Prion Disease Center “Prof. John Collinge” about Alzheimer’s disease infection in patients Said that Alzheimer’s infection (Alzheimer) does not have the same characteristics as a viral or bacterial outbreak such as the common cold or COVID-19. People who are likely to be infected by Alzheimer’s disease is a person who has had brain surgery using cells or tissues that has precursors to increase the chance of causingamnesia It is also a very rare event.
In addition, data from this research has shown us that Prion Disease is similar to Alzheimer’s disease, which is characterized by the spread of proteins throughout our brain system
Prion disease is caused by an abnormality that spreads in the brain. caused by protein infection And it often occurs naturally without any stimulation from external factors.
The figures reveal that at least 1,848 people with Alzheimer’s disease in the UK were given growth hormone between 1959 and 1985. That is extracted from the pituitary gland of a corpse or the body of a deceased person.
Even in 1985, after it emerged that some patients had died from a type of prion disease called Creutzfeldt-Jakob encephalopathy, Creutzfeldt-Jakob disease (CJD) resulted from the hormone used being contaminated with the protein that causes CJD. The treatment was eventually discontinued.
Read the full research here.
There have been 80 reported cases where amyloid-beta protein was found. (Amyloid-Beta) in the brain after death. But it is not yet clear whether there is a precursor to the development of Alzheimer’s disease. But other research indicates that We all may be at risk of developing Alzheimer’s disease if we are stimulated by it. Amyloid-beta is present in some hormones.
Researchers reported findings from eight people referred to the National Prion Clinic between 2017 and 2022.
All received cadaveric human growth hormone, but none had CJD. Five patients had dementia that met clinical criteria for Alzheimer’s disease. With onset at age 38, three patients had brain scans consistent with the diagnosis, while two had biomarkers meeting criteria for Alzheimer’s disease.
Of the other three patients, one had mild cognitive impairment. One person self-reported having intellectual difficulties. and have no such symptoms The first showed autopsy results consistent with Alzheimer’s disease. and the latter met the biomarker criteria for the disease.
Five patients had DNA data available, but only one showed genetic risk factors for late-onset Alzheimer’s disease. And no one knows that there is a genetic variation that causes early onset Alzheimer’s disease.
The researchers added that Patients show some symptoms that differ from typical symptoms of Alzheimer’s disease. This occurs naturally or is linked to genetic risk. It is assumed that this symptom may be caused by diseases of different origins or by different “strains” of Amyloid-beta
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Reference: www.theguardian.com
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