They discover a new molecule that promotes intestinal healing and reduces colorectal cancer

by time news

A⁣ team of ⁢researchers ⁤from the Karolinska Institute in ⁤Sweden has ‌identified a molecule that ​can help the ⁤intestine heal after damage, while at the same time suppressing the growth⁣ of⁣ tumors in⁢ the ⁣intestine. colorectal cancer. ‌This discovery lays the foundation for the development of new targeted therapies inflammatory ⁣bowel disease ​(IBD) and colon cancer.

Given the lack of response of many patients with inflammatory bowel diseases, such​ as Crohn’s disease or ulcerative​ colitisto the available‌ treatments, the need arises to look for​ new therapeutic ​strategies.‍ In a ‍study ⁢published in “Nature” magazine, Scientists suggest that promoting the regeneration ⁢of the⁤ intestinal ‍mucosa could be ⁤an effective alternative conventional immunosuppressive drugs.

However, this approach ⁣carries the risk of inducing uncontrolled tumor growth,⁢ as cancer cells can take advantage ⁢of the body’s natural healing processes.

The lead author of the study,⁣ Srustidhar ‌Das, group research ​specialist‌ led by Eduardo Villablanca at the Department of Medicine of the Karolinska Institute, highlights: “We​ have now identified a molecule that can help the‍ intestine ‍heal ⁣after ‍damage, while⁢ at the same time suppressing⁣ tumor growth in ‌colorectal cancer.”

Activation of​ the liver X receptor (LXR).

The ‍researchers found that ⁣the‍ activation ⁢of a protein called Liver X ⁤receptor (LXR) can promote ⁤tissue ‍regeneration and,⁣ in⁢ turn, inhibit tumor growth in colorectal cancer. Eduardo J.‍ Villablanca, professor at the Karolinska Institute and last author of the study, says:⁢ “The discovery⁤ of both functions was surprising. Now ​we ⁤need to study more closely how LXR⁢ controls tumor formation.”

Advanced technologies used in the study

To carry out this research, the team ‍used a number of cutting-edge techniques, such as intestinal‍ cell transcriptome‌ mapping and ​3D ⁤organoid culture, small, three-dimensional cellular structures that mimic the function and structure of organs ⁣on a miniature scale.

Furthermore, ⁣they‌ applied‌ spatial transcriptomics​ to⁤ map⁤ gene expression in different tissues, a methodology developed at SciLifeLab by scientists from the University ⁢of Royal Institute ⁣of⁤ Technology‌ (KTH) and the Karolinska Institute itself.

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How⁤ can new ⁣therapeutic​ strategies improve treatment​ for inflammatory bowel disease?

Interview Between Time.news Editor and Expert on Recent Discoveries⁤ in⁢ Gastrointestinal Health

Editor: Good morning, and welcome to Time.news. Today, we are fortunate ‌to ⁣have⁣ with⁤ us Dr. Srustidhar Das, a leading ⁣researcher from the Karolinska Institute in Sweden. Dr. Das, ⁤thank‍ you for joining us.

Dr. Das: Good morning! ‍Thank ​you for having me. It’s a pleasure to be ⁣here.

Editor: Your recent study ⁤published in⁤ “Nature” has generated quite a buzz in​ the medical community. Can ⁤you‍ tell us about the molecule⁣ your team identified and its potential impact on intestinal health?

Dr. Das: Certainly! ‌We discovered a ​specific molecule that shows ⁣promise in​ promoting the healing of the intestinal lining while simultaneously ⁢suppressing tumor growth associated with colorectal cancer. This dual action ⁣could significantly influence how we approach treatments for conditions like inflammatory bowel disease (IBD) and colon cancer.

Editor: That sounds groundbreaking!⁤ Many patients with ‍IBD, particularly​ those suffering from diseases like Crohn’s or ulcerative colitis,‍ don’t respond well to existing treatments. ⁤How ⁤does your‌ discovery offer new hope?

Dr. Das: Absolutely, the need for innovative therapeutic strategies has never been more apparent. Our findings suggest that enhancing‌ the regeneration of‍ the⁤ intestinal mucosa could serve as an effective alternative ‍to conventional immunosuppressive⁣ drugs, which often have limited effectiveness and undesirable side effects.

Editor: It’s great to ‌hear about alternative strategies, but ⁣you also mentioned concerns regarding uncontrolled tumor ​growth. Can you elaborate‍ on that?

Dr. Das: ⁢Yes, it’s a ⁢critical balance. ‍While ​promoting ⁢mucosal healing⁣ is essential,⁣ it also presents a risk; cancer cells can exploit the body’s natural healing processes for their advantage. Our⁣ team is working​ diligently​ to better understand how to harness ⁣this healing without inadvertently supporting tumorigenesis. ⁤

Editor: Conversations​ about balancing healing and ​cancer⁤ risk are crucial. What are the next steps in your research?

Dr. Das: We’re⁤ focused‌ on further ‍validating our results through preclinical studies. The ultimate goal is to ensure that we can safely target this new molecule ‍for therapeutic applications⁢ and assess ​its ⁣long-term safety and efficacy in human subjects.

Editor:‍ Fascinating! It seems like there’s a lot⁤ of potential for‍ this research. How do ⁣you see‌ the future of targeted therapies in this ‍field evolving?

Dr. Das: I believe we are on the cusp of a new era where ⁤precision medicine⁢ can play a significant‍ role in treating both IBD and colon cancer. By understanding individual patient profiles and how different‌ molecules‌ can ⁢work synergistically, we can‌ tailor treatments that are not only effective but​ also ‌minimize the risks associated with traditional therapies.

Editor: That’s a hopeful perspective! Before we wrap⁤ up, what message​ do you have ⁢for patients currently struggling with ⁢IBD ​and cancer?

Dr. Das: I⁤ want to emphasize that research is ongoing and rapidly advancing. If you’re struggling with these conditions, I encourage you to stay informed about new treatments ​and maintain ⁤open communication with your healthcare providers. There’s hope on the horizon, ⁤and we’re committed to finding solutions that truly⁤ make a difference.

Editor: Thank you,​ Dr. Das, for your ​insights and for your important ⁢work in this field. We‌ look forward to following your research ‍and the⁤ impact it will undoubtedly have​ on patients’ lives.

Dr. Das: Thank you for⁤ having me! It’s been a pleasure discussing this vital⁣ topic.

Editor: And‌ thank you ⁢to⁢ our audience‍ for​ tuning in to ‌Time.news. Stay informed with‍ us⁣ for more​ updates on health and science!

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